Genetic Defects monitored by RAAA

Arthrogryposis Multiplex (AM) Double Muscling (DM) Dwarfism (DW)
Contractural Arachnodactyly (CA) Hypotrichosis (HY) Alpha-Mannosidosis (MA)
Neuropathic Hydrocephalus (NH) Osteopetrosis (OS) Protoporphyria (PR)
Pulmonary Hypoplasia with Anasarca (PH) Syndactyly (SN) Tibial Hemimelia (TH)
Developmental Duplication (DD)    

No species is immune from genetic defects; several genetic defects have been discovered, and for the most part eliminated, in breeds of cattle. Due to their inheritance pattern, genetic defects can remain hidden for many generations, often not rearing their "ugly head" until some degree of inbreeding is practiced. Most genetic defects exist as simple recessives.

The majority of abnormal appearing calves are not genetic defects. Environmental effects such as the virus BVD and toxic weeds can mimic genetic defects. Therefore, extensive testing must be performed to rule out all non-genetic causes. Reference your RAAA Rules and Regulations (pg 17) to review the steps for investigating possible genetic defect carriers and a description of all genetic defects monitored by RAAA. Abnormal calves should be immediately reported to the National Office. It is imperative that the abnormal animal not be destroyed, as an autopsy must be performed and samples taken to verify parentage through DNA.